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Exposure to CSE from 3R4F, NF and ultralow nicotine cigarettes promotes the pro-inflammatory activation of BBB endothelial cells As shown in Figure 6A, endothelial cell expression of vascular endothelial adhesion molecule-1 (VCAM-1) was up-regulated following exposure to 3R4F, NF and ultralow nicotine CSEs, as compared to controls and 1R5F cigarette treated cultures

Discussion Tobacco use is an important global health issue

After exposure of HVSMCs to nicotine, the -actin mRNA level decreased by approximately 0.4-fold, whereas, the -actin mRNA level increased to approximately 1.7-fold after 48 h, respectively [33]

doi:10.3889/oamjms.2016.117 56