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Significantly, a recent in vitro study demonstrated that acute CSE induced increased infection severity in air-liquid interface cultures derived from human airway basal stem cells by impairing IFN responses and altering tissue repair (Purkayastha et al., 2020)

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Because e-cigarette exposure affected components in the lung associated with immune defense, the researchers wanted to test how e-cigarette-exposed mice would respond to flu infection

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