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doi: 10.1001/jama.281.24.2294 68
Originally posted 5 April 2017
Les fibres musculaires lisses qui entourent les bronches, contractes de faon chronique par les irritants du tabac, commencent se relcher
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Since IL-1 engages an IL-1 receptor on ILC3s and promotes their proliferation and IL-17A production 14 , these findings suggest that the smoking-induced sputum ILC3s and circulating memory-like CD4 + CD45RO + ILC3 subset in asthma patients could be generated by smoke-damaged airway epithelial cells rather than circulating immune cells
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