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Thus, the enhanced neurogenesis observed with IH EARLY could be mediated by upregulation of HIF1a in INPs, promoting the increase in the proportion of adult born neurons after the selective pressure of IH is removed

Early reports that Kupffer cell-induced oxidant stress and peroxynitrite formation may cause APAP-induced injury [92, 93] were questioned as the data could not be reproduced [9496], NADPH oxidase-deficient mice were not protected [97, 98] and mitochondria in centrilobular hepatocytes were identified as the main source of superoxide and peroxynitrite under these conditions [72, 99]