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A dual role of cyclin E in cell proliferation and apoptosis may provide a target for cancer therapy

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Alterations resulting from METex14 mutations are marked by the partial removal of the parallel membrane configuration or hindered attachment to the E3 ubiquitin-protein ligase CBL, which impedes MET protein ubiquitination and self-degradation, thereby extending the activation of subsequent signals [23, 24]

In research models, its studied for its interaction with GHRH receptors on the pituitary gland, where it prompts the pituitary to release endogenous growth hormone through the bodys own regulatory pathway rather than introducing growth hormone directly, as some other compounds in this research category do