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Protein kinase C and fatty acid transport protein 4 (FATP4) [144] play an important role in the increase of GLP-1 secretion and improvement in glycemic tolerance caused by oleic acid (OA), both in vivo and in vitro [145]

A systematic review suggested that insufficient cerebral perfusion may be a potential cause of cognitive dysfunction (Wang Y

The results, while concerning, confirm that NAION risk is rare but real for Ozempic users

Increasing the dose might work but adds risk

Common pathophysiological mechanisms of metabolic syndrome and NDD include insulin resistance of the brain, neuroinflammation, oxidative stress, dysfunction of the BBB, mitochondrial damage, dysbiosis of the intestinal microbiota and changes in the renin-angiotensin system