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By inhibiting autophagy, the TP53 mutation, which is less common in hematological cancers than in solid tumors, increases p53 expression and triggers apoptotic responses dependent on p53-upregulated modulator of apoptosis and BAX, leading to AML cell destruction (44)

Huang Q, Liu Z, Yang Y, Yang Y, Huang T, Hong Y, et al

Your body releases it from extracellular matrix proteins whenever tissue damage occurs, essentially functioning as a biological signal that activates repair mechanisms

Our bodies need these amino acids for optimal health

Effect sizes were considered to be small (Hedges g = 0.2, explaining 1% of the variance), intermediate (g = 0.5, explaining 9% of the variance) or large (g = 0.8, explaining 25% of the variance) according to 31