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Kishi et al., 2005

If the level of colchicine builds up too high, as it might if a usual dose is given to a patient with severe kidney disease, toxicity can occur, such as suppression of the production of blood cells

Meanwhile, knowing the mechanism of GSH-responsive and -exhausting cancer treatment strategies based on metal nanomedicines is vital for selecting an optimized approach to GSH consumption and improving the efficiency of cancer therapy

Downstream signaling from RTK activation: Receptor binding triggers autophosphorylation recruiting adaptor proteins initiating multiple pathways

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