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Clinical studies have shown that coagulation activation during ECMO initially leads to the deposition of fibrin, platelets, and leukocytes on the circuit surface (63), which can develop into sarcoid thrombosis in some patients (64)

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GSH reduction can occur in both (intrinsic) mitochondrial-mediated apoptosis and (extrinsic) death receptor-mediated apoptosis based on tumorigenesis by two possible mechanisms: gene mutation and transcription factors (TF)