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This post-translational modification is now recognised as a major mechanism of redox-regulated cell signalling, controlling the activity of transcription factors, kinases, phosphatases, and metabolic enzymes in response to the intracellular redox environment

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Wang et al., 2009, 2011), which could interact with behavioral measures of morphine tolerance

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Close observation of the presynaptic component alone has shown that the majority of putative presynaptic structures is stable and shows abundant expression of inhibitory pre and post synaptic proteins (Schuemann et al., 2013)