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Results In this study we monitored the intracellular amount of GSH and GSSG in the sensitive (CO23) and resistant strains to FLC or MFCG (CO23RFLC and CO23RFK) of C

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Nadeem et al., 2019)

Regarding non-GPX4-dependent pathways, a study on lung adenocarcinoma revealed that knockdown of nicotinamide adenine dinucleotide kinase (NADK) can suppress FSP1 expression by reducing NADPH levels, thereby enhancing the cytotoxicity of Erastin and RSL3, highlighting the critical role of the NADK-FSP1 axis in regulating ferroptosis (136)

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